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Thursday, June 11, 2015

Antibiotic Resistance Research: New strategies for stopping the spread of Staph and MRSA ♦ Physicists develop ultrasensitive nanomechanical biosenso ♦ Molecular docking site of a bacterial toxin identified

How bacteria survive antibiotics may improve treatment of infectious diseases Infectious diseases kill more people worldwide than any other single cause, but treatment often fails because a small fraction of bacterial cells can transiently survive antibiotics and recolonize the body. A study reveals that these so-called persisters form in response to adverse conditions through the action of a molecule called Obg, which plays an important role in all major cellular processes in multiple bacterial species.
New strategies for stopping the spread of Staph and MRSA Staphylococcus aureus -- better known as Staph -- is a common inhabitant of the human nose, and people who carry it are at increased risk for dangerous Staph infections. However, it may be possible to exclude these unwelcome guests using other more benign bacteria.
Molecular docking site of a bacterial toxin identified A team of pharmacologists and toxicologists have identified the molecular docking site that is responsible for the Clostridium difficile toxins being able to bind to its receptor on the membrane of the intestinal epithelium. This docking site functions like an elevator, transporting the toxins into the cell's interior.
Physicists develop ultra sensitive nano mechanical bio sensor Two young researchers have developed an ultra compact highly sensitive nano mechanical sensor for analyzing the chemical composition of substances and detecting biological objects, such as viral disease markers, which appear when the immune system responds to incurable or hard-to-cure diseases, including HIV, hepatitis, herpes, and many others. The sensor will enable doctors to identify tumor markers, whose presence in the body signals the emergence and growth of cancerous tumors;

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