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Friday, August 7, 2015

Cardiovascular Research:Get up for your heart health, move for your waistline ♦ Social factors linked to heart disease for all ♦ Why the human heart cannot regenerate

Get up for your heart health, move for your waistline More time spent standing rather than sitting could improve your blood sugar, fats in the blood and cholesterol levels. The study also shows that replacing time spent sitting with time walking could have additional benefits for your waistline and body mass index.
Byproduct of intestinal bacteria may jeopardize heart health in patients with kidney disease Blood levels of TMAO, a byproduct generated from intestinal bacterial as they metabolize dietary nutrients, progressively increase with advancing severity of kidney disease. TMAO levels are dramatically reduced when kidney function is restored following kidney transplantation, researchers say, noting that high TMAO levels are linked with an increased risk of atherosclerosis and premature death in patients with chronic kidney disease.
Social factors linked to heart disease for all The social determinants of health are multi-dimensional and multi-level, yet we have few studies that examine the social determinants in large, diverse populations. More extensive research and new interventions are needed, an expert writes, if we are to reach the a goal of increasing the proportion of the population in ideal cardiovascular health by 2020
Why the human heart cannot regenerate Damage to the human heart causes cardiac muscle cells to die, which in turn leads to reduced heart function and death. However, this is not the case for zebrafish or amphibians. If their hearts become damaged and cardiac muscle cells die, their remaining cardiac muscle cells can reproduce, allowing the heart to regenerate. Researchers have now found a possible explanation as to why this does not happen in humans.
Working to ensure the heart's ideal performance Utilizing a pharmaceutical treatment for systolic heart failure, that is being tested in clinical trials, new research determined the precise interaction between the drug and the cardiac myosin protein or the cardiac “motor,” forming a structure that regulates the contraction of cardiac muscle and allows the heart to efficiently pump oxygen-rich blood throughout the body.

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